MiR‐133 promotes cardiac reprogramming by directly repressing Snai1 and silencing fibroblast signatures-Reference-Cited by-同舟云学术

MiR‐133 promotes cardiac reprogramming by directly repressing Snai1 and silencing fibroblast signatures

Published:2014-06-11 Issue:14 Volume:33 Page:1565-1581
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Muraoka Naoto¹²,Yamakawa Hiroyuki¹²,Miyamoto Kazutaka¹²,Sadahiro Taketaro¹²,Umei Tomohiko¹,Isomi Mari¹,Nakashima Hanae¹,Akiyama Mizuha¹,Wada Rie¹,Inagawa Kohei¹²,Nishiyama Takahiko¹²,Kaneda Ruri¹²,Fukuda Toru³,Takeda Shu³,Tohyama Shugo²,Hashimoto Hisayuki²,Kawamura Yoshifumi⁴,Goshima Naoki⁵,Aeba Ryo⁶,Yamagishi Hiroyuki⁷,Fukuda Keiichi²,Ieda Masaki¹²⁸

Affiliation:

1. Department of Clinical and Molecular Cardiovascular Research Keio University School of Medicine Shinjuku‐ku Tokyo Japan

2. Department of Cardiology Keio University School of Medicine Shinjuku‐ku Tokyo Japan

3. Department of Physiology and Cell Biology Tokyo Medical and Dental University Bunkyo‐ku Tokyo Japan

4. Japan Biological Informatics Consortium (JBiC) Koto‐ku Tokyo Japan

5. Molecular Profiling Research Center for Drug Discovery National Institute of Advanced Industrial Science and Technology Koto‐ku Tokyo Japan

6. Division of Cardiovascular Surgery Keio University School of Medicine Shinjuku‐ku Tokyo Japan

7. Department of Pediatrics Keio University School of Medicine Shinjuku‐ku Tokyo Japan

8. JST CREST Shinjuku‐ku Tokyo Japan

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201387605

Reference28 articles.

1. Induced regeneration—the progress and promise of direct reprogramming for heart repair

2. Inefficient Reprogramming of Fibroblasts into Cardiomyocytes Using Gata4, Mef2c, and Tbx5

3. Direct Reprogramming of Human Fibroblasts toward a Cardiomyocyte-like State

4. Direct Reprogramming of Fibroblasts into Functional Cardiomyocytes by Defined Factors

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