BCL‐2‐family protein tBID can act as a BAX‐like effector of apoptosis-Reference-Cited by-同舟云学术

BCL‐2‐family protein tBID can act as a BAX‐like effector of apoptosis

Published:2021-12-21 Issue:2 Volume:41 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:The EMBO Journal

Author:

Flores‐Romero Hector¹²³^ORCID,Hohorst Lisa¹²^ORCID,John Malina³^ORCID,Albert Marie‐Christine⁴,King Louise E¹²,Beckmann Laura²⁵⁶,Szabo Tamas⁷,Hertlein Vanessa³,Luo Xu⁸⁹,Villunger Andreas⁷¹⁰¹¹^ORCID,Frenzel Lukas P²⁵⁶,Kashkar Hamid⁴^ORCID,Garcia‐Saez Ana J¹²³^ORCID

Affiliation:

1. Institute for Genetics University of Cologne Cologne Germany

2. Cologne Excellence Cluster on Cellular Stress Responses in Aging‐Associated Diseases (CECAD) University of Cologne Cologne Germany

3. Interfaculty Institute of Biochemistry Eberhard‐Karls‐Universität Tübingen Tübingen Germany

4. Institute for Molecular Immunology, and Center for Molecular Medicine Cologne (CMMC) Faculty of Medicine University Hospital of Cologne University of Cologne Cologne Germany

5. Department I of Internal Medicine University Hospital of Cologne Cologne Germany

6. Center of Integrated Oncology ABCD University Hospital of Cologne Cologne Germany

7. Division of Developmental Immunology Biocenter Medical University of Innsbruck Innsbruck Austria

8. Eppley Institute for Research in Cancer and Allied Diseases Fred & Pamela Buffett Cancer Center University of Nebraska Medical Center Omaha ME USA

9. Department of Pathology and Microbiology University of Nebraska Medical Center Omaha NE USA

10. CeMM Research Center for Molecular Medicine of the Austrian Academy of Sciences Vienna Austria

11. Ludwig Boltzmann Institute for Rare and Undiagnosed Diseases Vienna Austria

Funder

Deutsche Forschungsgemeinschaft

H2020 European Research Council

Austrian Science Fund

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2021108690

Reference76 articles.

1. BID ‐dependent release of mitochondrial SMAC dampens XIAP ‐mediated immunity against Shigella

2. Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations

3. Pro-apoptotic Cleavage Products of Bcl-xL Form Cytochrome c-conducting Pores in Pure Lipid Membranes

4. Bid: a Bax-like BH3 protein

5. Dynamic Interaction of cBid with Detergents, Liposomes and Mitochondria

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