Neuron‐specific translational control shift ensures proteostatic resilience during <scp>ER</scp> stress-Reference-Cited by-同舟云学术

Neuron‐specific translational control shift ensures proteostatic resilience during ER stress

Published:2022-07-06 Issue:16 Volume:41 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:The EMBO Journal

Author:

Wolzak Kimberly¹²^ORCID,Nölle Anna³,Farina Margherita¹,Abbink Truus EM⁴,van der Knaap Marjo S¹⁴,Verhage Matthijs¹²^ORCID,Scheper Wiep¹²^ORCID

Affiliation:

1. Department of Functional Genomics, Faculty of Science, Center for Neurogenomics and Cognitive Research (CNCR) Vrije Universiteit (VU) Amsterdam Amsterdam The Netherlands

2. Functional Genomics Section, Department of Human Genetics Amsterdam University Medical Centers (UMC) Location Vrije Universiteit Amsterdam The Netherlands

3. Department of Pathology Amsterdam University Medical Centers (UMC) Location Vrije Universiteit Amsterdam The Netherlands

4. Department of Child Neurology Amsterdam University Medical Centers (UMC) Location Vrije Universiteit Amsterdam The Netherlands

Funder

Alzheimer Nederland

Janssen Pharmaceuticals

ZonMw

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2021110501

Reference96 articles.

1. The heme-regulated inhibitor is a cytosolic sensor of protein misfolding that controls innate immune signaling

2. The switch-like expression of heme-regulated kinase 1 mediates neuronal proteostasis following proteasome inhibition

3. Anoxic induction of ATF-4 through HIF-1–independent pathways of protein stabilization in human cancer cells

4. ER and Nutrient Stress Promote Assembly of Respiratory Chain Supercomplexes through the PERK-eIF2α Axis

5. Neuronal development is promoted by weakened intrinsic antioxidant defences due to epigenetic repression of Nrf2

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