Suppressing toxic aggregates: let MIA do it!
Author:
Affiliation:
1. IMol Polish Academy of Sciences Warsaw Poland
2. Centre of New Technologies University of Warsaw Warsaw Poland
3. ReMedy International Research Agenda Unit University of Warsaw Warsaw Poland
Publisher
EMBO
Subject
General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience
Link
https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2021109001
Reference12 articles.
1. Mitochondrial protein-induced stress triggers a global adaptive transcriptional programme
2. Cytosolic Protein Vms1 Links Ribosome Quality Control to Mitochondrial and Cellular Homeostasis
3. Mitochondrial Stress Restores the Heat Shock Response and Prevents Proteostasis Collapse during Aging
4. Mitochondrial protein translocation-associated degradation
5. Cytosolic aggregation of mitochondrial proteins disrupts cellular homeostasis by stimulating the aggregation of other proteins;Nowicka U;bioRxiv,2021
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1. The emerging role of autophagy and mitophagy in tauopathies: From pathogenesis to translational implications in Alzheimer’s disease;Frontiers in Aging Neuroscience;2022-10-17
2. Long-term mitochondrial stress induces early steps of Tau aggregation by increasing reactive oxygen species levels and affecting cellular proteostasis;Molecular Biology of the Cell;2022-07-01
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