TRIM16 controls assembly and degradation of protein aggregates by modulating the p62‐NRF2 axis and autophagy-Reference-Cited by-同舟云学术

TRIM16 controls assembly and degradation of protein aggregates by modulating the p62‐NRF2 axis and autophagy

Published:2018-08-24 Issue:18 Volume:37 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Jena Kautilya Kumar¹²,Kolapalli Srinivasa Prasad¹,Mehto Subhash¹^ORCID,Nath Parej¹²,Das Biswajit³⁴,Sahoo Pradyumna Kumar¹,Ahad Abdul⁵,Syed Gulam Hussain⁶,Raghav Sunil K⁵,Senapati Shantibhusan³,Chauhan Swati⁷,Chauhan Santosh¹^ORCID

Affiliation:

1. Cell Biology and Infectious Diseases Unit Institute of Life Sciences Bhubaneswar India

2. School of Biotechnology KIIT University Bhubaneswar India

3. Tumor Microenvironment and Animal Models Institute of Life Sciences Bhubaneswar India

4. Manipal University Manipal India

5. Immuno‐Genomics and Systems Biology Institute of Life Sciences Bhubaneswar India

6. Molecular Virology and Infectious Diseases Institute of Life Sciences Bhubaneswar India

7. Translational Research Institute of Life Sciences Bhubaneswar India

Funder

DBT India Alliance

Science and Engineering Research Board

Department of Science and Technology, Ministry of Science and Technology

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201798358

Reference96 articles.

1. Protein quality control and elimination of protein waste: The role of the ubiquitin–proteasome system

2. Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death

3. Induction of autophagy by the MG-132 proteasome inhibitor is associated with endoplasmic reticulum stress in MCF-7 cells

4. TRIM16 Acts as an E3 Ubiquitin Ligase and Can Heterodimerize with Other TRIM Family Members

5. Protein Quality Control Acts on Folding Intermediates to Shape the Effects of Mutations on Organismal Fitness

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