Crosstalk between BRCA ‐ F anconi anemia and mismatch repair pathways prevents MSH 2‐dependent aberrant DNA damage responses-Reference-Cited by-同舟云学术

Crosstalk between BRCA ‐ F anconi anemia and mismatch repair pathways prevents MSH 2‐dependent aberrant DNA damage responses

Published:2014-06-26 Issue:15 Volume:33 Page:1698-1712
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Peng Min¹,Xie Jenny¹,Ucher Anna²,Stavnezer Janet²,Cantor Sharon B¹

Affiliation:

1. Department of Cancer Biology University of Massachusetts Medical School Women's Cancers Program UMASS Memorial Cancer Center Worcester MA USA

2. Department of Microbiology and Physiological Systems University of Massachusetts Medical School Women's Cancers Program UMASS Memorial Cancer Center Worcester MA USA

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201387530

Reference79 articles.

1. Preventing Nonhomologous End Joining Suppresses DNA Repair Defects of Fanconi Anemia

2. Saccharomyces cerevisiae MSH2, a mispaired base recognition protein, also recognizes Holliday junctions in DNA

3. Bypass of DNA Lesions Generated During Anticancer Treatment with Cisplatin by DNA Polymerase η

4. BRCA1, PARP, and 53BP1: conditional synthetic lethality and synthetic viability

5. Frameshift Mutation inPRKDC, the Gene for DNA-PKcs, in the DNA Repair-Defective, Human, Glioma-Derived Cell Line M059J

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