PRC 2 is dispensable for HOTAIR ‐mediated transcriptional repression
Author:
Affiliation:
1. Institut Curie PSL Research University Paris France
2. INSERM U934 CNRS UMR3215 Paris France
3. INSERM U900 Mines ParisTech Paris France
4. CNRS UMR 8015 Université Paris Descartes Paris France
Funder
Institut National Du Cancer
Agence Nationale de la Recherche
Cancéropôle Île-de-France
Seventh Framework Programme
Publisher
EMBO
Subject
General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience
Link
https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201695335
Reference50 articles.
1. Modeling of epigenome dynamics identifies transcription factors that mediate Polycomb targeting
2. The interaction of PRC2 with RNA or chromatin is mutually antagonistic
3. Noncoding RNA and Polycomb recruitment
4. Evf2 lncRNA/BRG1/DLX1 interactions reveal RNA-dependent chromatin remodeling inhibition
5. The Noncoding RNA Revolution—Trashing Old Rules to Forge New Ones
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