Complex IV subunit isoform COX 6A2 protects fast‐spiking interneurons from oxidative stress and supports their function-Reference-Cited by-同舟云学术

Complex IV subunit isoform COX 6A2 protects fast‐spiking interneurons from oxidative stress and supports their function

Published:2020-08-03 Issue:18 Volume:39 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Sanz‐Morello Berta¹,Pfisterer Ulrich¹,Winther Hansen Nikolaj²,Demharter Samuel¹,Thakur Ashish¹,Fujii Katsunori³,Levitskii Sergey A⁴,Montalant Alexia²,Korshunova Irina¹,Mammen Pradeep PA⁵,Kamenski Piotr⁴^ORCID,Noguchi Satoru⁶⁷,Aldana Blanca Irene⁸,Hougaard Karin Sørig⁹¹⁰,Perrier Jean‐François²^ORCID,Khodosevich Konstantin¹^ORCID

Affiliation:

1. Biotech Research and Innovation Centre (BRIC) Faculty of Health and Medical Sciences University of Copenhagen Copenhagen Denmark

2. Department of Neuroscience University of Copenhagen Copenhagen Denmark

3. Department of Pediatrics Chiba University Graduate School of Medicine Chiba Japan

4. Faculty of Biology Lomonosov Moscow State University Moscow Russia

5. Department of Internal Medicine University of Texas Southwestern Medical Center Dallas TX USA

6. Department of Neuromuscular Research National Institute of Neuroscience National Center of Neurology and Psychiatry Tokyo Japan

7. Medical Genome Center National Center of Neurology and Psychiatry Tokyo Japan

8. Department of Drug Design and Pharmacology University of Copenhagen Copenhagen Denmark

9. Section of Environmental Health National Research Centre for the Working Environment Copenhagen Denmark

10. Department of Public Health University of Copenhagen Copenhagen Denmark

Funder

Novo Nordisk Fonden

Lundbeckfonden

Det Frie Forskningsråd

Agnes og Poul Friis Fond

Russian Foundation for Fundamental Investigations

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2020105759

Reference80 articles.

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2. Ketamine-Induced Loss of Phenotype of Fast-Spiking Interneurons Is Mediated by NADPH-Oxidase

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5. Glutathione deficit during development induces anomalies in the rat anterior cingulate GABAergic neurons: Relevance to schizophrenia

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