Paternal USP26 mutations raise Klinefelter syndrome risk in the offspring of mice and humans-Reference-Cited by-同舟云学术

Paternal USP26 mutations raise Klinefelter syndrome risk in the offspring of mice and humans

Published:2021-05-12 Issue:13 Volume:40 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Liu Chao¹²^ORCID,Liu Hongbin³⁴,Zhang Haobo³⁴,Wang Lina¹⁵,Li Mengjing³⁴,Cai Feifei³,Wang Xiuge¹⁵,Wang Li³⁴,Zhang Ruidan¹⁵,Yang Sijie³⁴,Liu Wenwen¹⁵,Liang Yu⁶,Wang Liying¹⁵,Song Xiaohui³⁴,Su Shizhen³⁴,Gao Hui¹,Jiang Jing⁷,Li Jinsong⁷,Luo Mengcheng⁸,Gao Fei¹⁵^ORCID,Chen Qi⁹^ORCID,Li Wei¹²⁵^ORCID,Chen Zi‐Jiang³⁴⁶^ORCID

Affiliation:

1. State Key Laboratory of Stem Cell and Reproductive Biology Institute of Zoology, Stem Cell and Regenerative Medicine Innovation Institute Chinese Academy of Sciences Beijing China

2. Fertility Preservation Lab, Reproductive Medicine Center Guangdong Second Provincial General Hospital Guangzhou China

3. Center for Reproductive Medicine Cheeloo College of Medicine Key Laboratory of Reproductive Endocrinology of Ministry of Education Shandong University Jinan China

4. Shandong Key Laboratory of Reproductive Medicine Jinan China

5. University of Chinese Academy of Sciences Beijing China

6. Shanghai Key Laboratory for Assisted Reproduction and Reproductive Genetics Shanghai China

7. Genome Tagging Project (GTP) Center Shanghai Institute of Biochemistry and Cell Biology Center for Excellence in Molecular Cell Science Chinese Academy of Sciences Shanghai China

8. Department of Tissue and Embryology School of Basic Medical Sciences Wuhan University Wuhan China

9. Division of Biomedical Sciences School of Medicine University of California, Riverside Riverside CA USA

Funder

Youth Innovation Promotion Association of the Chinese Academy of Sciences

National Science Fund for Distinguished Young Scholars

National Natural Science Foundation of China

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2020106864

Reference56 articles.

1. Ensuring meiotic DNA break formation in the mouse pseudoautosomal region

2. ZIP4H (TEX11) Deficiency in the Mouse Impairs Meiotic Double Strand Break Repair and the Regulation of Crossing Over

3. Novel mutation in USP26 associated with azoospermia in a Sertoli cell‐only syndrome patient

4. Involvement of mouse Mlh1 in DNA mismatch repair and meiotic crossing over

5. ATM Promotes the Obligate XY Crossover and both Crossover Control and Chromosome Axis Integrity on Autosomes

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