USP49 negatively regulates tumorigenesis and chemoresistance through FKBP51‐AKT signaling-Reference-Cited by-同舟云学术

USP49 negatively regulates tumorigenesis and chemoresistance through FKBP51‐AKT signaling

Published:2017-03-31 Issue:10 Volume:36 Page:1434-1446
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Luo Kuntian¹²³,Li Yunhui¹²,Yin Yujiao¹²,Li Lei¹²,Wu Chenming¹²,Chen Yuping¹²,Nowsheen Somaira⁴,Hu Qi⁵,Zhang Lizhi⁶,Lou Zhenkun³^ORCID,Yuan Jian¹²³^ORCID

Affiliation:

1. Research Center for Translational MedicineEast Hospital Tongji University School of Medicine Shanghai China

2. Key Laboratory of Arrhythmias of the Ministry of Education of China East Hospital Tongji University School of Medicine Shanghai China

3. Department of Oncology Mayo Clinic Rochester MN USA

4. Medical Scientist Training Program Mayo Clinic School of Medicine Mayo Clinic Graduate School of Biomedical Sciences Rochester MN USA

5. Department of Biochemistry and Molecular Biology Mayo Clinic Rochester MN USA

6. Department of Laboratory Medicine and Pathology Mayo Clinic Rochester MN USA

Funder

National Natural Science Foundation of China

Mayo Clinic

National Cancer Institute

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201695669

Reference52 articles.

1. 3-Phosphoinositide-dependent protein kinase-1 (PDK1): structural and functional homology with the Drosophila DSTPK61 kinase

2. Implication of PI3K/Akt pathway in pancreatic cancer: When PI3K isoforms matter?

3. miR-190-Mediated Downregulation of PHLPP Contributes to Arsenic-Induced Akt Activation and Carcinogenesis

4. Activation of AKT Kinases in Cancer: Implications for Therapeutic Targeting

5. Ten years of protein kinase B signalling: a hard Akt to follow

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