AMPK promotes survival of c‐Myc‐positive melanoma cells by suppressing oxidative stress-Reference-Cited by-同舟云学术

AMPK promotes survival of c‐Myc‐positive melanoma cells by suppressing oxidative stress

Published:2018-02-12 Issue:5 Volume:37 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Kfoury Alain¹,Armaro Marzia¹,Collodet Caterina²³^ORCID,Sordet‐Dessimoz Jessica¹,Giner Maria Pilar²,Christen Stefan²,Moco Sofia²,Leleu Marion¹,Leval Laurence⁴,Koch Ute¹,Trumpp Andreas⁵⁶,Sakamoto Kei²³,Beermann Friedrich¹,Radtke Freddy¹^ORCID

Affiliation:

1. Ecole Polytechnique Fédérale de Lausanne School of Life Sciences Swiss Institute for Experimental Cancer Research Lausanne Switzerland

2. Nestlé Institute of Health Sciences SA Lausanne Switzerland

3. Ecole Polytechnique Fédérale de Lausanne School of Life Sciences Lausanne Switzerland

4. Institute of Pathology University Hospital Lausanne Lausanne Switzerland

5. Division of Stem Cells and Cancer Deutsches Krebsforschungszentrum (DKFZ) Heidelberg Germany

6. Heidelberg Institute for Stem Cell Technology and Experimental Medicine (HI‐STEM GmbH) Heidelberg Germany

Funder

Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung

Oncosuisse

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201797673

Reference57 articles.

1. Metastasizing Melanoma Formation Caused by Expression of Activated N-RasQ61K on an INK4a-Deficient Background

2. Melanoma genome sequencing reveals frequent PREX2 mutations

3. The landscape of somatic copy-number alteration across human cancers

4. Human melanoma-initiating cells express neural crest nerve growth factor receptor CD271

5. Benzimidazole derivative small-molecule 991 enhances AMPK activity and glucose uptake induced by AICAR or contraction in skeletal muscle

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