MicroRNA-22 Promotes Renal Tubulointerstitial Fibrosis by Targeting PTEN and Suppressing Autophagy in Diabetic Nephropathy
Author:
Affiliation:
1. Department of Pathophysiology, Guizhou Medical University, Guiyang, Guizhou 550025, China
2. Laboratory of Pathogenesis Research, Drug Prevention and Treatment of Major Diseases, Guizhou Medical University, Guiyang, Guizhou 550025, China
Abstract
Funder
Guiyang and Guizhou Medical University Joint Foundation
Publisher
Hindawi Limited
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
http://downloads.hindawi.com/journals/jdr/2018/4728645.pdf
Reference30 articles.
1. Transformation of interstitial fibroblasts and tubulointerstitial fibrosis in diabetic nephropathy
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3. Atg5-mediated autophagy deficiency in proximal tubules promotes cell cycle G2/M arrest and renal fibrosis
4. Podocyte Autophagy: A Potential Therapeutic Target to Prevent the Progression of Diabetic Nephropathy
5. miR-21 promotes renal fibrosis in diabetic nephropathy by targeting PTEN and SMAD7
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