Specificity of Antimicrobial Peptide LL-37 to Neutralize Periodontopathogenic Lipopolysaccharide Activity in Human Oral Fibroblasts
Author:
Publisher
Wiley
Subject
Periodontics
Link
http://www.joponline.org/doi/pdf/10.1902/jop.2012.110652
Reference44 articles.
1. Microbial etiological agents of destructive periodontal diseases
2. Lipopolysaccharide dependent cellular activation
3. Virulence factors of Actinobacillus actinomycetemcomitans
4. Virulence factors of Porphyromonas gingivalis
5. Lipopolysaccharides of Bacteroides intermedius (Prevotella intermedia) and Bacteroides (Porphyromonas) gingivalis induce interleukin-8 gene expression in human gingival fibroblast cultures
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1. The neutralising and stimulatory effects of antimicrobial peptide LL-37 in human gingival fibroblasts;Archives of Oral Biology;2023-04
2. Pathogenic Mechanisms of Fusobacterium nucleatum on Oral Epithelial Cells;Frontiers in Oral Health;2022-04-05
3. Cytocompatibility and Synergy of EGCG and Cationic Peptides Against Bacteria Related to Endodontic Infections, in Planktonic and Biofilm Conditions;Probiotics and Antimicrobial Proteins;2021-08-16
4. Effect of the Antimicrobial Peptide LL-37 on Gene Expression of Chemokines and 29 Toll-like Receptor-Associated Proteins in Human Gingival Fibroblasts Under Stimulation with Porphyromonas gingivalis Lipopolysaccharide;Probiotics and Antimicrobial Proteins;2019-11-04
5. Effects of LL-37 on Gingival Fibroblasts: A Role in Periodontal Tissue Remodeling?;Vaccines;2018-07-23
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