Human Monocyte Response to Cementum Extracts From Periodontally Diseased Teeth: Effect of Conditioning With Tetracycline
Author:
Publisher
Wiley
Subject
Periodontics
Link
http://www.joponline.org/doi/pdf/10.1902/jop.1996.67.7.682
Reference32 articles.
1. The Bacterial Etiology of Destructive Periodontal Disease: Current Concepts
2. The Role of the Host Response in Periodontal Disease Progression: Implications for Future Treatment Strategies
3. Inflammatory mediators and the destruction of bone
4. The role of inflammatory mediators in the pathogenesis of periodontal disease
5. Levels of interleukin 1beta in tissue from sites of active periodontal disease
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1. Tetracycline Conditioning Augments the In Vivo Inflammatory Response Induced by Cementum Extracts;Journal of Periodontology;2004-03
2. In Vitro Effect of Laser Irradiation on Cementum-Bound Endotoxin Isolated From Periodontally Diseased Roots;Journal of Periodontology;2002-11
3. Inhibition of Matrix Metalloproteinase-Mediated Periodontal Bone Loss in Rats: A Comparison of 6 Chemically Modified Tetracyclines;Journal of Periodontology;2002-07
4. Chlortetracycline modulates acute phase response of ex vivo perfused pig livers, and inhibits TNF-α secretion by isolated Kupffer cells;Comparative Immunology, Microbiology and Infectious Diseases;2001-04
5. Effects of Sex Hormones on Production of Interleukin-1 by Human Peripheral Monocytes;Journal of Periodontology;1999-07
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