Finding the needle in the haystack: BMP9 and 10 emerge from the genome in pulmonary arterial hypertension
Author:
Funder
British Heart Foundation
Publisher
European Respiratory Society (ERS)
Subject
Pulmonary and Respiratory Medicine
Reference23 articles.
1. Bone Morphogenetic Protein-9 Is a Circulating Vascular Quiescence Factor
2. Regulation of the ALK1 ligands, BMP9 and BMP10
3. Bone Morphogenetic Protein (BMP) and Activin Type II Receptors Balance BMP9 Signals Mediated by Activin Receptor-like Kinase-1 in Human Pulmonary Artery Endothelial Cells
4. Identification of BMP9 and BMP10 as functional activators of the orphan activin receptor-like kinase 1 (ALK1) in endothelial cells
5. Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension
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1. Circulating BMP10 Levels Associate With Late Postoperative Atrial Fibrillation and Left Atrial Endomysial Fibrosis;JACC: Clinical Electrophysiology;2024-07
2. Bone morphogenetic protein 10, a rising star in the field of diabetes and cardiovascular disease;Journal of Cellular and Molecular Medicine;2024-05
3. Seeing pulmonary hypertension through a paediatric lens: a viewpoint;European Respiratory Journal;2024-04-04
4. Human iPSCs as Model Systems for BMP-Related Rare Diseases;Cells;2023-09-02
5. GATA6 coordinates cross-talk between BMP10 and oxidative stress axis in pulmonary arterial hypertension;Scientific Reports;2023-04-22
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