Pim-1 Protects Retinal Ganglion Cells by Enhancing Their Regenerative Ability Following Optic Nerve Crush-Reference-Cited by-同舟云学术

Pim-1 Protects Retinal Ganglion Cells by Enhancing Their Regenerative Ability Following Optic Nerve Crush

Published:2020-06-30 Issue:3 Volume:29 Page:249-272
ISSN:1226-2560
Container-title:Experimental Neurobiology
language:en
Short-container-title:Exp Neurobiol

Author:

Zhang Shoumei¹²,Shuai Li³,Wang Dong¹,Huang Tingting¹,Yang Shengsheng⁴,Miao Mingyong⁴,Liu Fang¹,Xu Jiajun¹

Affiliation:

1. Department of Anatomy, Second Military Medical University, Shanghai 200433, China

2. Translational Medical Center for Stem Cell Therapy, Shanghai East Hospital, Tongji University School of Medicine, Shanghai 200120, China

3. Department of Health Administration, Second Military Medical University, Shanghai 200433, China

4. Department of Biochemistry and Molecular Biology, Second Military Medical University, Shanghai 200433, China

Funder

National Natural Science Foundation of China

Publisher

The Korean Society for Brain and Neural Science

Subject

Cellular and Molecular Neuroscience,Clinical Neurology

Link

https://pdf.medrang.co.kr/EN/2020/029/En029-03-06.pdf

Reference79 articles.

1. Quantitative iTRAQ Analysis of Retinal Ganglion Cell Degeneration after Optic Nerve Crush

2. Optic nerve crush: protection and regeneration

3. Fischer D, Pavlidis M, Thanos S (2000) Cataractogenic lens injury prevents traumatic ganglion cell death and promotes axonal regeneration both in vivo and in culture. Invest Ophthalmol Vis Sci 41: 3943-3954

4. Apoptotic Retinal Ganglion Cell Death in an Autoimmune Glaucoma Model Is Accompanied by Antibody Depositions

5. B-RAF kinase drives developmental axon growth and promotes axon regeneration in the injured mature CNS

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