Mitochondrial Dysfunction of Immortalized Human Adipose Tissue-Derived Mesenchymal Stromal Cells from Patients with Parkinson’s Disease-Reference-Cited by-同舟云学术

Mitochondrial Dysfunction of Immortalized Human Adipose Tissue-Derived Mesenchymal Stromal Cells from Patients with Parkinson’s Disease

Published:2013-12-30 Issue:4 Volume:22 Page:283-300
ISSN:1226-2560
Container-title:Experimental Neurobiology
language:en
Short-container-title:Exp Neurobiol

Author:

Moon Hyo Eun¹²³,Yoon Seung Hee⁴,Hur Yong Suk⁵,Park Hyung Woo¹²³,Ha Ji Young⁴,Kim Kyung-Hee⁴,Shim Jung Hee⁴,Yoo Seung Hyun⁵,Son Jin H.⁴,Paek Seung Leal¹²³⁶,Kim In Keyoung¹,Hwang Jae Ha¹,Kim Dong Gyu¹,Kim Han-Joon⁷,Jeon Beom Seok⁷,Park Sung Sup⁸,Paek Sun Ha¹²³

Affiliation:

1. Department of Neurosurgery, Seoul National University College of Medicine, Seoul 110-744, Korea

2. Cancer Research Institute, Seoul National University College of Medicine, Seoul 110-744, Korea

3. Ischemic/Hypoxic Disease Institute, Seoul National University College of Medicine, Seoul 110-744, Korea

4. Department of Brain & Cognitive Sciences, College of Pharmacy, Brain Disease Research Institute, Ewha Woman's University, Seoul 120-750, Korea

5. Department of Biochemistry, Inha University School of Medicine, Incheon 402-751, Korea

6. Department of Neurosurgery, Mayo Clinic, USA

7. Department of Neurology, Seoul National University College of Medicine, Seoul 110-744, Korea

8. Department of Laboratory Medicine, Seoul National University Hospital, Seoul 110-744, Korea

Funder

Korea Institute of Planning & Evaluation for Technology in Food, Agriculture, Forestry, and Fisheries

Korea Healthcare Technology R&D Project, Ministry of Health & Welfare

Publisher

The Korean Society for Brain and Neural Science

Subject

Cellular and Molecular Neuroscience,Clinical Neurology

Link

https://synapse.koreamed.org/pdf/10.5607/en.2013.22.4.283

Reference44 articles.

1. Parkinson patient fibroblasts show increased alpha-synuclein expression

2. Mitochondrial biology and oxidative stress in Parkinson disease pathogenesis

3. Expanding insights of mitochondrial dysfunction in Parkinson's disease

4. The antioxidant Trolox helps recovery from the familial Parkinson's disease-specific mitochondrial deficits caused by PINK1- and DJ-1-deficiency in dopaminergic neuronal cells

5. Mitochondrial Dysfunction and Oxidative Damage in parkin-deficient Mice

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