4-hydroxy-2(E)-Nonenal facilitates NMDA-Induced Neurotoxicity via Triggering Mitochondrial Permeability Transition Pore Opening and Mitochondrial Calcium Overload-Reference-Cited by-同舟云学术

4-hydroxy-2(E)-Nonenal facilitates NMDA-Induced Neurotoxicity via Triggering Mitochondrial Permeability Transition Pore Opening and Mitochondrial Calcium Overload

Published:2013-09-30 Issue:3 Volume:22 Page:200-207
ISSN:1226-2560
Container-title:Experimental Neurobiology
language:en
Short-container-title:Exp Neurobiol

Author:

Choi In-Young¹,Lim Ji-Hyae²,Kim Chunsook³,Song Hwa Young¹,Ju Chung¹,Kim Won-Ki¹

Affiliation:

1. Department of Neuroscience, College of Medicine, Korea University, Seoul 136-705, Korea

2. Laboratory of Medical Genetics, Medical Research Institute, Cheil General Hospital and Women's Healthcare Center, Seoul 100-280, Korea

3. Department of Nursing, Andong Science College, Andong 760-709, Korea

Funder

ministry of health & welfare

Bio & Medical Technology Development Program

Publisher

The Korean Society for Brain and Neural Science

Subject

Cellular and Molecular Neuroscience,Clinical Neurology

Link

https://synapse.koreamed.org/pdf/10.5607/en.2013.22.3.200

Reference33 articles.

1. Calcium in Ischemic Cell Death

2. Calcium, mitochondria and oxidative stress in neuronal pathology. Novel aspects of an enduring theme

3. Mitochondrial contributions to tissue damage in stroke

4. Expanding insights of mitochondrial dysfunction in Parkinson's disease

5. Mitochondria and Ca2+in cell physiology and pathophysiology

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