PINK1 Deficiency Decreases Expression Levels of mir-326, mir-330, and mir-3099 during Brain Development and Neural Stem Cell Differentiation-Reference-Cited by-同舟云学术

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PINK1 Deficiency Decreases Expression Levels of mir-326, mir-330, and mir-3099 during Brain Development and Neural Stem Cell Differentiation

Published:2016-02-29 Issue:1 Volume:25 Page:14-23
ISSN:1226-2560
Container-title:Experimental Neurobiology
language:en
Short-container-title:Exp Neurobiol

Author:

Choi Insup¹²,Woo Joo Hong²,Jou Ilo¹²³,Joe Eun-hye¹²³⁴⁵

Affiliation:

1. Neuroscience Graduate Program Department of Biomedical Sciences, Ajou University School of Medicine, Suwon 16499, Korea

2. Chronic Inflammatory Disease Research Center, Ajou University School of Medicine, Suwon 16499, Korea

3. Department of Pharmacology, Ajou University School of Medicine, Suwon 16499, Korea

4. Department of Brain Science, Ajou University School of Medicine, Suwon 16499, Korea

5. Brain Disease Research Center, Ajou University School of Medicine, Suwon 16499, Korea

Funder

National Research Foundation of Korea

Publisher

The Korean Society for Brain and Neural Science

Subject

Cellular and Molecular Neuroscience,Neurology (clinical)

Link

http://synapse.koreamed.org/pdf/10.5607/en.2016.25.1.14

Reference50 articles.

1. Localization of a Novel Locus for Autosomal Recessive Early-Onset Parkinsonism, PARK6, on Human Chromosome 1p35-p36

2. Wild-type PINK1 Prevents Basal and Induced Neuronal Apoptosis, a Protective Effect Abrogated by Parkinson Disease-related Mutations

3. Mutations in PTEN-induced putative kinase 1 associated with recessive parkinsonism have differential effects on protein stability

4. PINK1 Defect Causes Mitochondrial Dysfunction, Proteasomal Deficit and α-Synuclein Aggregation in Cell Culture Models of Parkinson's Disease

5. C-terminal truncation and Parkinson's disease-associated mutations down-regulate the protein serine/threonine kinase activity of PTEN-induced kinase-1

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