Inactivation of Aconitase by Tetrahydrobiopterin in DArgic Cells: Relevance to PD
Author:
Affiliation:
1. Department of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, Seoul 138-736, Korea
Publisher
The Korean Society for Brain and Neural Science
Subject
Cellular and Molecular Neuroscience,Neurology (clinical)
Link
https://synapse.koreamed.org/pdf/10.5607/en.2010.19.1.23
Reference27 articles.
1. Dopamine Oxidation Alters Mitochondrial Respiration and Induces Permeability Transition in Brain Mitochondria
2. Oxidative Inactivation of Mitochondrial Aconitase Results in Iron and H2O2-Mediated Neurotoxicity in Rat Primary Mesencephalic Cultures
3. Tetrahydrobiopterin Is Released from and Causes Preferential Death of Catecholaminergic Cells by Oxidative Stress
4. Dopamine-dependent cytotoxicity of tetrahydrobiopterin: a possible mechanism for selective neurodegeneration in Parkinson's disease
5. Involvement of apoptosis and calcium mobilization in tetrahydrobiopterin-induced dopaminergic cell death
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