Vitamin E inhibits hepatic oxidative stress, toxicity and hyperproliferation in rats treated with the renal carcinogen ferric nitrilotriacetate
Author:
Publisher
Informa UK Limited
Subject
Biochemistry (medical),Cell Biology,Clinical Biochemistry,Physiology,Biochemistry
Link
http://www.tandfonline.com/doi/pdf/10.1179/135100005X21642
Reference48 articles.
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2. Free radicals, antioxidant enzymes, and carcinogenesis
3. Reactive Oxygen Species and Antioxidants in Signal Transduction and Gene Expression
4. Effect of dietary vitamin E on ferric nitrilotriacetate-induced nephrotoxicity in rats
5. Knekt P, Aroma A, Maatela J et al. Vitamin E and cancer prevention.Am J Clin Nutr1991; 53: 283S-286S.
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1. Hepatoprotective Potential of Aqueous Extract of Hibiscus rosasinensis and Butea monosperma against Fe-NTA induced Hepatotoxicity in Rats;Research Journal of Pharmacy and Technology;2022-07-29
2. Synthesis of Vitamin E Novel Analogues as Anti-Cancer Compounds;Jundishapur Journal of Natural Pharmaceutical Products;2016-01-25
3. Assessment of genotoxicity associated with Behcet’s disease using sister-chromatid exchange assay: vitamin E versus mitomycin C;Cytotechnology;2014-05-23
4. Ameliorative effect of polyphenols fromPadina boergeseniiagainst ferric nitrilotriacetate induced renal oxidative damage: With inhibition of oxidative hemolysis andin vitrofree radicals;Environmental Toxicology;2014-01-24
5. Vitamin E acetate addition to poly(d,l)lactic acid modifies its mechanical behavior without affecting biocompatibility;Journal of Applied Polymer Science;2013-09-30
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