Functional neural stem cell isolation from brains of adult mutant SOD1 (SOD1G93A) transgenic amyotrophic lateral sclerosis (ALS) mice
Author:
Publisher
Informa UK Limited
Subject
Clinical Neurology,Neurology,General Medicine
Link
http://www.tandfonline.com/doi/pdf/10.1179/016164110X12807570509899
Reference21 articles.
1. Amyotrophic lateral sclerosis: A review of current concepts
2. ALS-Linked SOD1 Mutant G85R Mediates Damage to Astrocytes and Promotes Rapidly Progressive Disease with SOD1-Containing Inclusions
3. Impaired Spinal Cord Glutamate Transport Capacity and Reduced Sensitivity to Riluzole in a Transgenic Superoxide Dismutase Mutant Rat Model of Amyotrophic Lateral Sclerosis
4. Motor Neuron Degeneration in Mice that Express a Human Cu,Zn Superoxide Dismutase Mutation
5. Rats Expressing Human Cytosolic Copper–Zinc Superoxide Dismutase Transgenes with Amyotrophic Lateral Sclerosis: Associated Mutations Develop Motor Neuron Disease
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1. Oxidized SOD1 accelerates cellular senescence in neural stem cells;Stem Cell Research & Therapy;2024-02-27
2. Adult Neurogenesis in Health and Disease;Neural Stem Cells and Adult Neurogenesis;2019
3. CRISPR/Cas9-mediated targeted gene correction in amyotrophic lateral sclerosis patient iPSCs;Protein & Cell;2017-04-11
4. Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1G93A mouse model of amyotrophic lateral sclerosis;Journal of Chemical Neuroanatomy;2016-10
5. Adult neurogenesis and neurodegenerative diseases: A systems biology perspective;American Journal of Medical Genetics Part B: Neuropsychiatric Genetics;2016-02-16
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