Contribution of hexose-6-phosphate dehydrogenase to NADPH content and redox environment in the endoplasmic reticulum
Author:
Publisher
Informa UK Limited
Subject
Biochemistry, medical,Cell Biology,Clinical Biochemistry,Physiology,Biochemistry
Link
http://www.tandfonline.com/doi/pdf/10.1179/174329210X12650506623249
Reference27 articles.
1. Enhanced Glutathione Levels and Oxidoresistance Mediated by Increased Glucose-6-phosphate Dehydrogenase Expression
2. Failure to increase glucose consumption through the pentose-phosphate pathway results in the death of glucose-6-phosphate dehydrogenase gene-deleted mouse embryonic stem cells subjected to oxidative stress
3. Glucose-6-phosphate dehydrogenase plays a crucial role in protection from redox-stress-induced apoptosis
4. Direct protein–protein interaction of 11β-hydroxysteroid dehydrogenase type 1 and hexose-6-phosphate dehydrogenase in the endoplasmic reticulum lumen
5. Evidence That the 11 β-Hydroxysteroid Dehydrogenase (11 β-HSD1) Is Regulated by Pentose Pathway Flux
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