Reproductive Toxicity of T Cells in Early Life: Abnormal Immune Development and Postnatal Diseases-Reference-Cited by-同舟云学术

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Reproductive Toxicity of T Cells in Early Life: Abnormal Immune Development and Postnatal Diseases

Published:2017-07-10 Issue:10 Volume:18 Page:
ISSN:1389-4501
Container-title:Current Drug Targets
language:en
Short-container-title:CDT

Author:

Liu Han-xiao¹,Jiang Aifang²,Chen Ting¹,Qu Wen¹,Yan Hui-yi¹,Ping Jie³

Affiliation:

1. Department of Pharmacology, Wuhan University School of Basic Medical Sciences, Wuhan 430071, China

2. Clinical Center of Reproductive Medicine, Affiliated Hospital of Weifang Medical University, Weifang 261031, China

3. Department of Pharmacology, Wuhan University School of Basic Medical Sciences, 185, Donghu Road, Wuhan 430071, China

Publisher

Bentham Science Publishers Ltd.

Subject

Clinical Biochemistry,Drug Discovery,Pharmacology,Molecular Medicine

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Attenuated cholesterol metabolism pathway suppresses regulatory T cell development in prenatal nicotine exposed female mice;Toxicology;2019-12

2. Inhibition of thymocyte autophagy-associated CD4+T thymopoiesis is involved in asthma susceptibility in mice exposed to caffeine prenatally;Archives of Toxicology;2019-02-25

3. α7 nAChR mediated Fas demethylation contributes to prenatal nicotine exposure-induced programmed thymocyte apoptosis in mice;Oncotarget;2017-10-05

4. Editorial: Drug Targets and Biomarkers for Obstetric/Gynecologic/Reproductive Diseases;Current Drug Targets;2017-07-10

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