Involvement of Advanced Glycation End-products (AGEs) in Alzheimers Disease-Reference-Cited by-同舟云学术

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Involvement of Advanced Glycation End-products (AGEs) in Alzheimers Disease

Published:2004-02-01 Issue:1 Volume:1 Page:39-46
ISSN:1567-2050
Container-title:Current Alzheimer Research
language:en
Short-container-title:CAR

Author:

Takeuchi Masayoshi,Kikuchi Seiji,Sasaki Nobuyuki,Suzuki Takako,Watai Takayuki,Iwaki Mina,Bucala Richard,Yamagishi Sho-ichi

Publisher

Bentham Science Publishers Ltd.

Subject

Clinical Neurology,Neurology

Cited by 115 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Microvascular disease and its association with dementia in patients with type 2 diabetes: A nationwide cohort study in Taiwan;Diabetes, Obesity and Metabolism;2024-08-29

2. PVP-AuNP Impedes Glycation Mediated Hen Egg White Lysozyme Aggregation Under Physiological Condition;BioNanoScience;2024-04-17

3. PVP-AuNP impedes glycation mediated Hen Egg White Lysozyme aggregation under physiological condition;2024-02-12

4. HIF-1α serves as a co-linker between AD and T2DM;Biomedicine & Pharmacotherapy;2024-02

5. Toxic advanced glycation end-products (TAGE) are major structures of cytotoxic AGEs derived from glyceraldehyde;Medical Hypotheses;2024-02

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