Research progress on the function and mechanism of CXorf67 in PFA ependymoma
Author:
Publisher
Science China Press., Co. Ltd.
Subject
Multidisciplinary
Link
https://engine.scichina.com/doi/pdf/A1E3A7EE8ECD44E09F85D38992F4FAAF
Reference29 articles.
1. Pajtler K W, Wen J, Sill M. Molecular heterogeneity and CXorf67 alterations in posterior fossa group A (PFA) ependymomas. Acta Neuropathol, 2018, 136: 211-226.
2. Hubner J M, Muller T, Papageorgiou D N, et al. EZHIP/CXorf67 mimics K27M mutated oncohistones and functions as an intrinsic inhibitor of PRC2 function in aggressive posterior fossa ependymoma. Neuro Oncol, 2019, 21: 878–889.
3. Dewaele B, Przybyl J, Quattrone A. Identification of a novel, recurrent MBTD1-CXorf67 fusion in low-grade endometrial stromal sarcoma. Int J Cancer, 2014, 134: 1112-1122.
4. Pajtler K W, Pfister S M, Kool M. Molecular dissection of ependymomas. Oncoscience, 2015, 2: 827-828.
5. Louis D N, Perry A, Reifenberger G. The 2016 World Health Organization classification of tumors of the central nervous system: A summary. Acta Neuropathol, 2016, 131: 803-820.
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1. Synergistic effect of poly (ADP-ribose) polymerase (PARP) inhibitor with chemotherapy on CXorf67-elevated posterior fossa group A ependymoma;Chinese Medical Journal;2024-03-13
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