Structural and functional studies of PR-DUB: molecular mechanism of its specific H2AK119 deubiquitination activity
Author:
Publisher
Science China Press., Co. Ltd.
Subject
General Medicine
Link
https://engine.scichina.com/doi/pdf/4EBB2F0DC87E427FA59B6FF5C486C09E
Reference13 articles.
1. Schuettengruber B, Bourbon H M, Di Croce L. Genome regulation by polycomb and trithorax: 70 years and counting. Cell, 2017, 171: 34-57.
2. Zhao X, Wu X. Polycomb-group proteins in the initiation and progression of cancer. J Genet Genomics, 2021, 48: 433-443.
3. Barbour H, Daou S, Hendzel M. Polycomb group-mediated histone H2A monoubiquitination in epigenome regulation and nuclear processes. Nat Commun, 2020, 11:
4. Kolovos P, Nishimura K, Sankar A. PR-DUB maintains the expression of critical genes through FOXK1/2- and ASXL1/2/3-dependent recruitment to chromatin and H2AK119ub1 deubiquitination. Genome Res, 2020, 30: 1119-1130.
5. Li R, Huang D, Zhao Y. PR-DUB safeguards Polycomb repression through H2AK119ub1 restriction. Cell Prolif, 2023, doi: 10.1111/cpr.13457:
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