SMC4 attenuation induces cancer cells to enter a diapause like state: Synergistic regulation of lactylation and cytoplasmic division
Author:
Publisher
Science China Press., Co. Ltd.
Subject
Multidisciplinary
Link
https://engine.scichina.com/doi/pdf/1CBDDE58CD64484D8F1E3F7C8F81EB54
Reference9 articles.
1. Dhimolea E, de Matos Simoes R, Kansara D. An embryonic diapause-like adaptation with suppressed myc activity enables tumor treatment persistence. Cancer Cell, 2021, 39: 240-256.e11.
2. Rehman S K, Haynes J, Collignon E. Colorectal cancer cells enter a diapause-like dtp state to survive chemotherapy. Cell, 2021, 184: 226-242.e21.
3. Wang Q, Wang C, Li N. Condensin Smc4 promotes inflammatory innate immune response by epigenetically enhancing NEMO transcription. J Autoimmunity, 2018, 92: 67-76.
4. Baergen A K, Jeusset L M, Lichtensztejn Z. Diminished condensin gene expression drives chromosome instability that may contribute to colorectal cancer pathogenesis. Cancers, 2019, 11:
5. Sun X, He L, Liu H. The diapause-like colorectal cancer cells induced by SMC4 attenuation are characterized by low proliferation and chemotherapy insensitivity. Cell Metab, 2023, 35: 1563-1579.e8.
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