THE RELATIONSHIP BETWEEN CENTRAL BLOOD FLOW AND MICROCIRCULATION WITH THE AMOUNT OF ELASTIC COMPRESSION IN POST-THROMBOPHLEBOTIC SYNDROME IN THE STAGE OF DECOMPENSATION

Abstract

Summary. Aim. To determine the influence of elastic compression on the venous and arterial velocity of the main blood flow of the lower limb and the skin microcirculation of the anterior part of the foot in patients with a decompensated form of postthrombophlebotic syndrome. Materials and methods. 21 patient with post-thrombophlebotic syndrome in the stage of decompensation participated in the study. Among them were 12 (57 %) women and 9 (43 %) men. The average age of the patients was (58.5±6.4) years. All subjects of the study were measured for the ankle-brachial pressure index, deep femoral-popliteal index, regional perfusion index, tcpO2, tcpCO2, arterial blood flow velocity in the femoral artery, and venous blood flow velocity distal to the saphenofemoral junction. After the measurements, the following measurements were performed after using elastic medical knitwear of different compression classes. Research results. With I class compression, the venous blood flow rate was (0.064±0.007) (p=0.0001). The venous velocity of the femoral blood flow significantly decreased, on average, by 7.4 times compared to the initial conditions when using class IV compression (p=0.0001). values of tcpCO2 increased significantly already at the 1st compression class. (p=0.0001) and continued to increase until the end of the study. Analyzing the changes in tcpO2 and tcpCO2, it was established that at the 3rd compression class, there was a cross between the oxygen and carbon dioxide tension indicators, which was accompanied by the predominance of tcpCO2 pressure over tcpO2 at the next increase in the compression class. Conclusions. The arterial blood flow rate decreased by 8.5 times compared to the input data at the maximum pressure of the fourth compression class (p=0.0001). Venous velocity of blood flow in the femoral vein at the maximum pressure of elastic compression decreased venous velocity by 7.4 times compared to the initial normal (p=0.0001). tcpCO2 significantly increases at compression of 15 mmHg (p=0.0001).