Upregulation of Heme Oxygenase-1 in an Animal Model of Takotsubo Cardiomyopathy-Reference-Cited by-同舟云学术

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Upregulation of Heme Oxygenase-1 in an Animal Model of Takotsubo Cardiomyopathy

Published:2009 Issue:6 Volume:73 Page:1141-1146
ISSN:1346-9843
Container-title:Circulation Journal
language:en
Short-container-title:Circ J

Author:

Ueyama Takashi¹,Kawabe Tetsuya²,Hano Takuzo²,Tsuruo Yoshihiro¹,Ueda Kazuki³,Ichinose Masao³,Kimura Hiroko⁴,Yoshida Ken-ichi⁵

Affiliation:

1. Department of Anatomy and Cell Biology, Wakayama Medical University School of Medicine

2. Center for Educational Research and Development, Wakayama Medical University School of Medicine

3. Second Department of Internal Medicine, Wakayama Medical University School of Medicine

4. Department of Forensic Medicine, Juntendo University School of Medicine

5. Department of Legal Medicine, Graduate School of Medicine, University of Tokyo

Publisher

Japanese Circulation Society

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Link

http://www.jstage.jst.go.jp/article/circj/73/6/73_CJ-08-0988/_pdf

Reference30 articles.

1. Stress (Takotsubo) cardiomyopathy—a novel pathophysiological hypothesis to explain catecholamine-induced acute myocardial stunning

2. 2. Akashi YJ, Goldstein DS, Barbaro G, Ueyama T. Takotsubo cardiomyopathy: A new form of acute, reversible heart failure. Circulation 2008; 118: 2754-2762.

3. 3. Kume T, Kawamoto T, Okura H, Toyota E, Neishi Y, Watanabe N, et al. Local release of catecholamines from the hearts of patients with tako-tsubo-like left ventricular dysfunction. Circ J 2008; 72: 106-108.

4. Microcirculation and oxidative stress

5. 5. Dhalla KS, Rupp H, Beamish RE, Dhalla NS. Mechanisms of alterations in cardiac membrane Ca²⁺ transport due to excess catecholamines. Cardiovasc Drugs Ther 1996; 10: 231-238.

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