Effect of Dipeptidyl Peptidase-4 Inhibitors on Cardiovascular Outcome and Cardiac Function in Patients With Diabetes and Heart Failure　― Insights From the Ibaraki Cardiac Assessment Study-Heart Failure (ICAS-HF) Registry ―-Reference-Cited by-同舟云学术

Effect of Dipeptidyl Peptidase-4 Inhibitors on Cardiovascular Outcome and Cardiac Function in Patients With Diabetes and Heart Failure　― Insights From the Ibaraki Cardiac Assessment Study-Heart Failure (ICAS-HF) Registry ―

Published:2017 Issue:11 Volume:81 Page:1662-1669
ISSN:1346-9843
Container-title:Circulation Journal
language:en
Short-container-title:Circ J

Author:

Yamamoto Masayoshi¹,Seo Yoshihiro¹,Ishizu Tomoko¹,Nishi Isao²,Hamada-Harimura Yoshie¹,Machino-Ohtsuka Tomoko¹,Sato Kimi¹,Sai Seika¹,Sugano Akinori¹,Obara Kenichi³,Aonuma Kazutaka¹

Affiliation:

1. Cardiovascular Division, Institute of Clinical Medicine, Graduate School of Comprehensive Human Sciences, University of Tsukuba

2. Department of Cardiology, Tsuchiura Clinical Education and Training Center, University of Tsukuba Hospital

3. Division of Cardiology, Ryugasaki Saiseikai General Hospital

Publisher

Japanese Circulation Society

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Link

https://www.jstage.jst.go.jp/article/circj/81/11/81_CJ-17-0240/_pdf

Reference27 articles.

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2. 2. Nichols GA, Gullion CM, Koro CF, Ephross SA, Brown JB. The incidence of congestive heart failure in type 2 diabetes. Diabetes Care 2005; 27: 1879–1884.

3. 3. Pacheco BP, Crajoinas RO, Couto GK, Davel AP, Lessa LM, Rossoni LV, et al. Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats. J Hypertens 2011; 29: 520–528.

4. 4. Gomez N, Touihri K, Matheeussen V, Mendes Da Costa A, Mahmoudabady M, Mathieu M, et al. Dipeptidyl peptidase IV inhibition improves cardiorenal function in overpacing-induced heart failure. Eur J Heart Fail 2012; 14: 14–21.

5. 5. Esposito G, Cappetta D, Russo R, Rivellino A, Ciuffreda LP, Roviezzo F, et al. Sitagliptin reduces inflammation, fibrosis and preserves diastolic function in a rat model of heart failure with preserved ejection fraction. Br J Pharmacol, doi:10.1111/bph.13686.

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