Endothelial Dysfunction The First Step Toward Coronary Arteriosclerosis

Author:

Vanhoutte Paul M.1

Affiliation:

1. Department of Pharmacology, Li Ka Shing Faculty of Medicine, The University of Hong Kong

Publisher

Japanese Circulation Society

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Reference43 articles.

1. 1. Furchgott RF, Zawadzki JV. The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine. Nature 1980; 288: 373-376.

2. 2. Furchgott RF, Vanhoutte PM. Endothelium-derived relaxing and contracting factors. FASEB 1989; J3: 2007-2017.

3. 3. Luscher TF, Vanhoutte PM. The Endothelium: Modulator of Cardiovascular Function. Boca Raton: CRC Press, Inc; 1990; 1-228.

4. 4. Furchgott RF. Studies on relaxation of rabbit aorta by sodium nitrite: The basis for the proposal that acid-activatable inhibitory factor from bovine retractor penis is inorganic nitrite and the endothelium-derived relaxing factor is nitric oxide. In: Vanhoutte PM, editor. Vasodilatation: Vascular Smooth Muscle, Peptides, Autonomic Nerves and Endothelium. New York: Raven Press; 1988; 401-414.

5. 5. Ignarro LJ, Byrns RE, Wood KS. Biochemical and pharmacological properties of endothelium-derived relaxing factor and its similarity to nitric oxide radical. In: Vanhoutte, PM, editor. Vasodilatation: Vascular Smooth Muscle, Peptides, Autonomic Nerves and Endothelium. New York: Raven Press; 1988; 427-436.

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