Ca2+ Overload Evokes a Transient Outward Current in Guinea-Pig Ventricular Myocytes
Author:
Affiliation:
1. Second Department of Internal Medicine, Akita University School of Medicine
Publisher
Japanese Circulation Society
Subject
Cardiology and Cardiovascular Medicine,General Medicine
Link
https://www.jstage.jst.go.jp/article/circj/66/1/66_1_87/_pdf
Reference34 articles.
1. 1. Furukawa T, Myerburg RJ, Furukawa N, Bassett AL, Kimura S. Differences in transient outward currents of feline endocardial and epicardial myocytes. Circ Res 1990; 67: 1287-1291.
2. 2. Rozanski GJ, Xu Z, Zhang K, Patel KP. Alterd K+ current of ventricular myocytes in rats with chronic myocardial infarction. Am J Physiol 1998; 274: H259-H265.
3. 3. Miyashita T, Kubota I, Yamaki M, Watanabe T, Yamauti S, Tomoike H. 4-Aminopyridine inhibits the occurrence of ventricular fibrillation but not ventricular tachycardia in the reperfused, isolated rat heart. Jpn Circ J 2000; 64: 602-605.
4. 4. Kawano S, Hirayama Y, Hiraoka M. Activation mechanism of Ca2+-sensitive transient outward current in rabbit ventricular myocytes. J Physiol 1995; 486: 593-604.
5. 5. Sipido KR, Callewaert G, Carmeliet E. [Ca2+]i transients and [Ca2+]i-dependent chloride current in single Purkinje cells from rabbit heart. J Physiol 1993; 468: 641-667.
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