CC Chemokine Receptor-2 Deficiency Attenuates Oxidative Stress and Infarct Size Caused by Myocardial Ischemia-Reperfusion in Mice-Reference-Cited by-同舟云学术

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CC Chemokine Receptor-2 Deficiency Attenuates Oxidative Stress and Infarct Size Caused by Myocardial Ischemia-Reperfusion in Mice

Published:2006 Issue:3 Volume:70 Page:342-351
ISSN:1346-9843
Container-title:Circulation Journal
language:en
Short-container-title:Circ J

Author:

Hayasaki Takanori¹,Kaikita Koichi¹,Okuma Toshiyuki²,Yamamoto Eiichiro¹,Kuziel William A.³,Ogawa Hisao¹,Takeya Motohiro²

Affiliation:

1. Department of Cardiovascular Medicine, Graduate School of Medical Sciences, Kumamoto University

2. Department of Cell Pathology, Graduate School of Medical Sciences, Kumamoto University

3. Autoimmune and Inflammatory Diseases, Protein Design Labs Inc

Publisher

Japanese Circulation Society

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Link

http://www.jstage.jst.go.jp/article/circj/70/3/70_3_342/_pdf

Reference49 articles.

1. Myocardial reperfusion, limitation of infarct size, reduction of left ventricular dysfunction, and improved survival. Should the paradigm be expanded?

2. Postreperfusion inflammation: a model for reaction to injury in cardiovascular disease

3. Induction of apoptosis in myocardial infarction and its possible relationship to nitric oxide synthase in macrophages

4. Chemokines — Chemotactic Cytokines That Mediate Inflammation

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1. An injectable polyacrylamide/chitosan-based hydrogel with highly adhesive, stretchable and electroconductive properties loaded with irbesartan for treatment of myocardial ischemia-reperfusion injury;International Journal of Biological Macromolecules;2024-05

2. Macrophages in cardiac remodelling after myocardial infarction;Nature Reviews Cardiology;2023-01-10

3. Macrophages in myocardial infarction;American Journal of Physiology-Cell Physiology;2022-10-01

4. Reperfusion Cardiac Injury: Receptors and the Signaling Mechanisms;Current Cardiology Reviews;2022-09

5. Role of CCR2-Positive Macrophages in Pathological Ventricular Remodelling;Biomedicines;2022-03-12

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