Cardioprotective Effect of Aprotinin on Myocardial Ischemia/Reperfusion Injury During Cardiopulmonary Bypass
Author:
Affiliation:
1. Siyami Ersek Thoracic and Cardiovascular Surgery Center, Department of Anesthesia and Reanimation
2. Siyami Ersek Thoracic and Cardiovascular Surgery Center, Department of Cardiovascular Surgery
Publisher
Japanese Circulation Society
Subject
Cardiology and Cardiovascular Medicine,General Medicine
Link
http://www.jstage.jst.go.jp/article/circj/70/11/70_11_1432/_pdf
Reference31 articles.
1. Myocardial infarct extension during reperfusion after coronary artery occlusion: Pathologic evidence
2. Role of endothelial dysfunction in the pathogenesis of reperfusion injury after myocardial ischemia
3. Lower cardiac troponin T levels in patients undergoing cardiopulmonary bypass and receiving high-dose aprotinin therapy indicate reduction of perioperative myocardial damage
4. Aprotinin Reduces Nitric Oxide Production in Vitro and in Vivo in a Dose-Dependent Manner
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3. Long noncoding RNA-MEG3 contributes to myocardial ischemia–reperfusion injury through suppression of miR-7-5p expression;Bioscience Reports;2019-08
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