Differential Myolysis of Myocardium and Skeletal Muscle in Hamsters With Dilated Cardiomyopathy Beneficial Protective Effect of Diltiazem-Reference-Cited by-同舟云学术

Differential Myolysis of Myocardium and Skeletal Muscle in Hamsters With Dilated Cardiomyopathy Beneficial Protective Effect of Diltiazem

Published:2006 Issue:11 Volume:70 Page:1497-1502
ISSN:1346-9843
Container-title:Circulation Journal
language:en
Short-container-title:Circ J

Author:

Kato Yosuke¹²,Iwase Mitsunori¹³,Takagi Kenji¹,Nishizawa Takao⁴,Kanazawa Hiroaki⁵,Matsushita Aya¹,Umeda Hisashi³,Izawa Hideo⁴,Noda Akiko¹,Koike Yasuo¹,Nagata Kohzo¹,Yokota Mitsuhiro⁶

Affiliation:

1. Pathophysiological Laboratory Sciences, Graduate School of Medicine, Nagoya University

2. Department of Pathobiological Science and Technology, School of Health Science, Faculty of Medicine, Tottori University

3. Division of Integrated Medicine and Cardiology, Toyota Memorial Hospital

4. Department of Cardiology, Graduate School of Medicine, Nagoya University

5. School of Nursing, University of Shizuoka

6. Department of Genome Science, School of Dentistry, Aichi-Gakuin University

Publisher

Japanese Circulation Society

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Link

http://www.jstage.jst.go.jp/article/circj/70/11/70_11_1497/_pdf

Reference19 articles.

1. The causes of dilated cardiomyopathy: A clinicopathologic review of 673 consecutive patients

2. Mutations in the human δ-sarcoglycan gene in familial and sporadic dilated cardiomyopathy

3. Autosomal recessive limbgirdle muscular dystrophy, LGMD2F, is caused by a mutation in the δ–sarcoglycan gene

4. Both hypertrophic and dilated cardiomyopathies are caused by mutation of the same gene, -sarcoglycan, in hamster: An animal model of disrupted dystrophin-associated glycoprotein complex

5. VEGF-mediated angiogenesis is impaired by angiotensin type 1 receptor blockade in cardiomyopathic hamster hearts

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1. Activation of Toll-like receptor 3 amplifies mesenchymal stem cell trophic factors and enhances therapeutic potency;American Journal of Physiology-Cell Physiology;2012-11-15

2. Developmental Changes in the ECG of a Hamster Model of Muscular Dystrophy and Heart Failure;Frontiers in Pharmacology;2012

3. Gait Disturbances in Dystrophic Hamsters;Journal of Biomedicine and Biotechnology;2011

4. Activation of host tissue trophic factors through JAK-STAT3 signaling: a mechanism of mesenchymal stem cell-mediated cardiac repair;American Journal of Physiology-Heart and Circulatory Physiology;2010-11

5. Ca2+overload and mitochondrial permeability transition pore activation in living δ-sarcoglycan-deficient cardiomyocytes;American Journal of Physiology-Cell Physiology;2010-09