Nicorandil Improves Post-Ischemic Myocardial Dysfunction in Association With Opening the Mitochondrial K<sub>ATP</sub> Channels and Decreasing Hydroxyl Radicals in Isolated Rat Hearts-Reference-Cited by-同舟云学术

Nicorandil Improves Post-Ischemic Myocardial Dysfunction in Association With Opening the Mitochondrial K_ATP Channels and Decreasing Hydroxyl Radicals in Isolated Rat Hearts

Published:2006 Issue:12 Volume:70 Page:1650-1654
ISSN:1346-9843
Container-title:Circulation Journal
language:en
Short-container-title:Circ J

Author:

Lu Chuanjiang¹,Minatoguchi Shinya¹,Arai Masazumi¹,Wang Ningyuan¹,Chen Xue-Hai¹,Bao Narentuoya¹,Kawamura Itta¹,Yasuda Shinji¹,Kobayashi Hiroyuki¹,Wu Der-Jinn²,Takemura Genzou¹,Fujiwara Hisayoshi¹

Affiliation:

1. Second Department of Internal Medicine, Gifu University School of Medicine

2. Chung Shang Medical University Hospital

Publisher

Japanese Circulation Society

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Link

https://www.jstage.jst.go.jp/article/circj/70/12/70_12_1650/_pdf

Reference34 articles.

1. 1. Garlid KD, Paucek P, Yarov-Yarovoy V, Murray HN, Darbenzio RB, D'Alonzo AJ, et al. Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K⁺ channels: Possible mechanism of cardioprotection. Circ Res 1997; 81: 1072-1082.

2. 2. Kim JH, Jeong MH, Yun KH, Kim KH, Kang DK, Hong SN, et al. Myocardial protective effects on nicorandil during percutaneous coronary intervention in patients with unstable angina. Circ J 2005; 69: 306-310.