A Natural p300-Specific Histone Acetyltransferase Inhibitor, Curcumin, in Addition to Angiotensin-Converting Enzyme Inhibitor, Exerts Beneficial Effects on Left Ventricular Systolic Function After Myocardial Infarction in Rats-Reference-Cited by-同舟云学术

A Natural p300-Specific Histone Acetyltransferase Inhibitor, Curcumin, in Addition to Angiotensin-Converting Enzyme Inhibitor, Exerts Beneficial Effects on Left Ventricular Systolic Function After Myocardial Infarction in Rats

Published:2011 Issue:9 Volume:75 Page:2151-2159
ISSN:1346-9843
Container-title:Circulation Journal
language:en
Short-container-title:Circ J

Author:

Sunagawa Yoichi¹²³,Morimoto Tatsuya³,Wada Hiromichi²,Takaya Tomohide⁴²,Katanasaka Yasufumi³,Kawamura Teruhisa²,Yanagi Shigeki⁵,Marui Akira⁵,Sakata Ryuzo⁵,Shimatsu Akira⁶,Kimura Takeshi⁴,Kakeya Hideaki⁷,Fujita Masatoshi¹,Hasegawa Koji²

Affiliation:

1. Department of Human Health Sciences, Graduate School of Medicine, Kyoto University

2. Division of Translational Research, Kyoto Medical Center, National Hospital Organization

3. Division of Molecular Medicine, School of Pharmaceutical Sciences, University of Shizuoka

4. Department of Cardiovascular Medicine, Graduate School of Medicine, Kyoto University

5. Department of Cardiovascular Surgery, Graduate School of Medicine, Kyoto University

6. Clinical Research Institute, Kyoto Medical Center, National Hospital Organization

7. Department of System Chemotherapy and Molecular Sciences, Division of Bioinformatics and Chemical Genomics, Graduate School of Pharmaceutical Sciences, Kyoto University

Publisher

Japanese Circulation Society

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Link

http://www.jstage.jst.go.jp/article/circj/75/9/75_CJ-10-1072/_pdf

Reference40 articles.

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2. 3. Francis GS, Chu C. Compensatory and maladaptive responses to cardiac dysfunction. Curr Opin Cardiol 1994; 9: 280-288.

3. 4. Katz AM. Maladaptive growth in the failing heart: The cardiomyopathy of overload. Cardiovasc Drugs Ther 2002; 16: 245-249.

4. THE CELLULAR AND MOLECULAR RESPONSE OF CARDIAC MYOCYTES TO MECHANICAL STRESS

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