Enhancement of Nitric Oxide Production Is Responsible for Minimal Intimal Hyperplasia of Autogenous Rabbit Arterial Grafts

Author:

Tabata Koki12,Komori Kimihiro2,Otsuka Ryo1,Kajikuri Junko1,Itoh Takeo1

Affiliation:

1. Department of Pharmacology, Graduate School of Medical Sciences, Nagoya City University

2. Division of Vascular Surgery, Department of Surgery, Nagoya University Graduate School of Medicine

Publisher

Japanese Circulation Society

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Reference34 articles.

1. 1. Furchgott RF, Zawadzki JV. The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine. Nature 1980; 288: 373–376.

2. 2. Martin W, Villani GM, Jothianandan D, Furchgott RF. Depression of contractile responses in rat aorta by spontaneously released endothelium-derived relaxing factor. J Pharmacol Exp Ther 1986; 237: 529–538.

3. 3. Komori K, Suzuki H. Electrical responses of smooth muscle cells during cholinergic vasodilation in the rabbit saphenous artery. Circ Res 1987; 61: 586–593.

4. 4. Félétou M, Vanhoutte PM. Endothelium-derived hyperpolarizing factor: Where are we now? Arterioscler Thromb Vasc Biol 2006; 26: 1215–1225.

5. 5. Kuriyama H, Kitamura K, Itoh T, Inoue R. Physiological features of visceral smooth muscle cells, with special reference to receptors and ion channels. Physiol Rev 1998; 78: 811–920.

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