Binge Alcohol Exposure Triggers Atrial Fibrillation Through T-Type Ca2+ Channel Upregulation via Protein Kinase C (PKC) / Glycogen Synthesis Kinase 3β (GSK3β) / Nuclear Factor of Activated T-Cells (NFAT) Signaling ― An Experimental Account of Holiday Heart Syndrome ―
Author:
Affiliation:
1. Department of Pathophysiology, Oita University School of Medicine
2. Department of Clinical Examination and Diagnostics, Oita University School of Medicine
3. Montreal Heart Institute Research Center, University of Montreal
Publisher
Japanese Circulation Society
Subject
Cardiology and Cardiovascular Medicine,General Medicine
Link
https://www.jstage.jst.go.jp/article/circj/84/11/84_CJ-20-0288/_pdf
Reference36 articles.
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3. 3. Taufiq F, Maharani N, Li P, Kurata Y, Ikeda N, Kuwabara M, et al. Uric acid-induced enhancements of Kv1.5 protein expression and channel activity via the Akt-HSF1-Hsp70 pathway in HL-1 atrial myocytes. Circ J 2019; 83: 718–726.
4. 4. Sano F, Ohira T, Kitamura A, Imano H, Cui R, Kiyama M, et al. Heavy alcohol consumption and risk of atrial fibrillation: The Circulatory Risk in Communities Study (CIRCS). Circ J 2014; 78: 955–961.
5. 5. Tonelo D, Providência R, Gonçalves L. Holiday heart syndrome revisited after 34 years. Arq Bras Cardiol 2013; 101: 183–189.
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