Administration of Recombinant Soluble Urokinase Receptor Per Se Is Not Sufficient to Induce Podocyte Alterations and Proteinuria in Mice-Reference-Cited by-同舟云学术

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Administration of Recombinant Soluble Urokinase Receptor Per Se Is Not Sufficient to Induce Podocyte Alterations and Proteinuria in Mice

Published:2014-05-01 Issue:8 Volume:25 Page:1662-1668
ISSN:1046-6673
Container-title:Journal of the American Society of Nephrology
language:en
Short-container-title:JASN

Author:

Cathelin Dominique,Placier Sandrine,Ploug Michael,Verpont Marie-Christine,Vandermeersch Sophie,Luque Yosu,Hertig Alexandre,Rondeau Eric,Mesnard Laurent

Publisher

American Society of Nephrology (ASN)

Subject

Nephrology,General Medicine

Reference43 articles.

1. Recurrence of idiopathic nephrotic syndrome after renal transplantation.;Hoyer;Lancet,1972

2. The Buffalo/Mna rat, an animal model of FSGS recurrence after renal transplantation

3. Modification of kidney barrier function by the urokinase receptor

4. Circulating urokinase receptor as a cause of focal segmental glomerulosclerosis

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