Constitutively Active SPAK Causes Hyperkalemia by Activating NCC and Remodeling Distal Tubules
Author:
Publisher
American Society of Nephrology (ASN)
Subject
Nephrology,General Medicine
Reference53 articles.
1. Activation of the renal Na+:Cl- cotransporter by angiotensin II is a WNK4-dependent process
2. The absence of intrarenal ACE protects against hypertension
3. WNK4 is the major WNK positively regulating NCC in the mouse kidney
4. Angiotensin II induces phosphorylation of the thiazide-sensitive sodium chloride cotransporter independent of aldosterone
5. Increasing plasma [K+] by intravenous potassium infusion reduces NCC phosphorylation and drives kaliuresis and natriuresis
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