Role of the Akt/FoxO3a Pathway in TGF-β1–Mediated Mesangial Cell Dysfunction: A Novel Mechanism Related to Diabetic Kidney Disease-Reference-Cited by-同舟云学术

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Role of the Akt/FoxO3a Pathway in TGF-β1–Mediated Mesangial Cell Dysfunction: A Novel Mechanism Related to Diabetic Kidney Disease

Published:2006-11-02 Issue:12 Volume:17 Page:3325-3335
ISSN:1046-6673
Container-title:Journal of the American Society of Nephrology
language:en
Short-container-title:JASN

Author:

Kato Mitsuo,Yuan Hang,Xu Zhong-Gao,Lanting Linda,Li Shu-Lian,Wang Mei,Hu Mickey C.-T.,Reddy Marpadga A.,Natarajan Rama

Publisher

American Society of Nephrology (ASN)

Subject

Nephrology,General Medicine

Reference50 articles.

1. Mechanisms of Glucose-Enhanced Extracellular Matrix Accumulation in Rat Glomerular Mesangial Cells

2. Early increased renal procollagen α1(IV) mRNA levels in streptozotocin induced diabetes

3. Gene expression of metalloproteinase and its inhibitor in mesangial cells exposed to high glucose

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