Genetic Variants of the COL4A3, COL4A4, and COL4A5 Genes Contribute to Thinned Glomerular Basement Membrane Lesions in Sporadic IgA Nephropathy Patients-Reference-Cited by-同舟云学术

Genetic Variants of the COL4A3, COL4A4, and COL4A5 Genes Contribute to Thinned Glomerular Basement Membrane Lesions in Sporadic IgA Nephropathy Patients

Published:2022-10-05 Issue:1 Volume:34 Page:132-144
ISSN:1046-6673
Container-title:Journal of the American Society of Nephrology
language:en
Short-container-title:JASN

Author:

Yuan Xiaohan,Su Qing,Wang Hui,Shi Sufang,Liu Lijun,Lv Jicheng,Wang Suxia,Zhu Li,Zhang Hong

Funder

National Key Research and Development Program of China

National Science Foundation for Excellent Young Scholars

National Science Foundation of China

Natural Science Foundation of Beijing Municipality

National High Level Hospital Clinical Research Funding

CAMS Innovation Fund for Medical Sciences

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Nephrology,General Medicine

Reference30 articles.

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2. The level of galactose-deficient IgA1 in the sera of patients with IgA nephropathy is associated with disease progression;Zhao;Kidney Int,2012

3. Aberrant IgA1 glycosylation is inherited in familial and sporadic IgA nephropathy;Gharavi;J Am Soc Nephrol,2008

4. Aberrant galactosylation of IgA1 is involved in the genetic susceptibility of Chinese patients with IgA nephropathy;Lin;Nephrol Dial Transplant,2009

5. Ultrastructural changes of glomerular basement membrane in IgA nephritis: Relationship to hematuria;Taguchi;Ultrastruct Pathol,1988

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