Loss of the Ciliary KinaseNek8Causes Left-Right Asymmetry Defects-Reference-Cited by-同舟云学术

Loss of the Ciliary KinaseNek8Causes Left-Right Asymmetry Defects

Published:2012-12-28 Issue:1 Volume:24 Page:100-112
ISSN:1046-6673
Container-title:Journal of the American Society of Nephrology
language:en
Short-container-title:JASN

Author:

Manning Danielle K.,Sergeev Mikhail,van Heesbeen Roy G.,Wong Michael D.,Oh Jin-Hee,Liu Yan,Henkelman R. Mark,Drummond Iain,Shah Jagesh V.,Beier David R.

Publisher

American Society of Nephrology (ASN)

Subject

Nephrology,General Medicine

Reference73 articles.

1. Chapter 6 Mouse Models of Polycystic Kidney Disease

2. The Polycystic Kidney Disease-1 Protein, Polycystin-1, Binds and Activates Heterotrimeric G-Proteinsin Vitro

3. Polycystin-2, the protein mutated in autosomal dominant polycystic kidney disease (ADPKD), is a Ca2+-permeable nonselective cation channel

4. Polycystin-2 Is a Novel Cation Channel Implicated in Defective Intracellular Ca2+ Homeostasis in Polycystic Kidney Disease

5. Constitutive Activation of G-proteins by Polycystin-1 Is Antagonized by Polycystin-2

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