p53 Is Renoprotective after Ischemic Kidney Injury by Reducing Inflammation-Reference-Cited by-同舟云学术

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p53 Is Renoprotective after Ischemic Kidney Injury by Reducing Inflammation

Published:2012-12-06 Issue:1 Volume:24 Page:113-124
ISSN:1046-6673
Container-title:Journal of the American Society of Nephrology
language:en
Short-container-title:JASN

Author:

Sutton Timothy A.,Hato Takashi,Mai Erik,Yoshimoto Momoko,Kuehl Sarah,Anderson Melissa,Mang Henry,Plotkin Zoya,Chan Rebecca J.,Dagher Pierre C.

Publisher

American Society of Nephrology (ASN)

Subject

Nephrology,General Medicine

Reference32 articles.

1. Cellular pathophysiology of ischemic acute kidney injury

2. Pathophysiology of ischemic acute kidney injury

3. Acute Renal Failure in Critically Ill PatientsA Multinational, Multicenter Study

4. Acute kidney injury: what's the prognosis?

5. Clinical Consequences of Acute Kidney Injury

Cited by 84 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Aquaporin 1 is renoprotective in septic acute kidney injury by attenuating inflammation, apoptosis and fibrosis through inhibition of P53 expression;Frontiers in Immunology;2024-08-22

2. Cell death induced by acute renal injury: a perspective on the contributions of accidental and programmed cell death;American Journal of Physiology-Renal Physiology;2024-07-01

3. Energy depletion by cell proliferation sensitizes the kidney epithelial cells to injury;American Journal of Physiology-Renal Physiology;2024-03-01

4. TNF-α from the Proximal Nephron Exacerbates Aristolochic Acid Nephropathy;Kidney360;2023-11-21

5. Aquaporin 1 Facilitates Ferroptosis, M1 Polarization, Mitochondrial Dysfunction, and Autophagy Damage on Lipopolysaccharide-Induced Macrophage Through Down-Regulation of P53 Signaling Pathway;DNA and Cell Biology;2023-08-01

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