T Cells in Crescentic Glomerulonephritis
Author:
Publisher
American Society of Nephrology (ASN)
Subject
Nephrology,General Medicine
Reference115 articles.
1. Evidence for delayed-type hypersensitivity mechanisms in glomerular crescent formation
2. Antibody independent crescentic glomerulonephritis in μ chain deficient mice
3. Susceptibility to anti-glomerular basement membrane disease and Goodpasture syndrome is linked to MHC class II genes and the emergence of T cell-mediated immunity in mice.
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1. Mucosal-associated invariant T cells contribute to suppression of inflammatory myeloid cells in immune-mediated kidney disease;Nature Communications;2023-11-15
2. Three Diseases Mediated by Different Immunopathologic Mechanisms—ANCA-Associated Vasculitis, Anti-Glomerular Basement Membrane Disease, and Immune Complex-Mediated Glomerulonephritis—A Common Clinical and Histopathologic Picture: Rapidly Progressive Crescentic Glomerulonephritis;Biomedicines;2023-11-06
3. Tr1 Cells Emerge and Suppress Effector Th17 Cells in Glomerulonephritis;The Journal of Immunology;2023-10-18
4. EGF-Receptor–Dependent TLR7 Signaling in Macrophages Promotes Glomerular Injury in Crescentic Glomerulonephritis;Laboratory Investigation;2023-09
5. Stimulation of Immune Checkpoint Molecule B and T-Lymphocyte Attenuator Alleviates Experimental Crescentic Glomerulonephritis;Journal of the American Society of Nephrology;2023-06-26
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