Lack of Arginine Vasopressin–Induced Phosphorylation of Aquaporin-2 Mutant AQP2-R254L Explains Dominant Nephrogenic Diabetes Insipidus-Reference-Cited by-同舟云学术

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Lack of Arginine Vasopressin–Induced Phosphorylation of Aquaporin-2 Mutant AQP2-R254L Explains Dominant Nephrogenic Diabetes Insipidus

Published:2005-08-24 Issue:10 Volume:16 Page:2872-2880
ISSN:1046-6673
Container-title:Journal of the American Society of Nephrology
language:en
Short-container-title:JASN

Author:

de Mattia Fabrizio,Savelkoul Paul J.M.,Kamsteeg Erik-Jan,Konings Irene B.M.,van der Sluijs Peter,Mallmann Rudolf,Oksche Alexander,Deen Peter M.T.

Publisher

American Society of Nephrology (ASN)

Subject

Nephrology,General Medicine

Reference38 articles.

1. Nephrogenic diabetes insipidus

2. Vasopressin increases water permeability of kidney collecting duct by inducing translocation of aquaporin-CD water channels to plasma membrane.

3. Requirement of Human Renal Water Channel Aquaporin-2 for Vasopressin-dependent Concentration of Urine

4. New mutations in the AQP2 gene in nephrogenic diabetes insipidus resulting in functional but misrouted water channels.

5. An aquaporin-2 water channel mutant which causes autosomal dominant nephrogenic diabetes insipidus is retained in the Golgi complex.

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1. Tubuloid differentiation to model the human distal nephron and collecting duct in health and disease;Cell Reports;2024-01

2. Aquaporin Gating: A New Twist to Unravel Permeation through Water Channels;International Journal of Molecular Sciences;2022-10-14

3. Deciphering the role of aquaporins in metabolic diseases: A mini review;The American Journal of the Medical Sciences;2022-08

4. Exploratory phosphoproteomics profiling of Aedes aegypti Malpighian tubules during blood meal processing reveals dramatic transition in function;PLOS ONE;2022-07-08

5. Nephrogenic diabetes insipidus: a comprehensive overview;Journal of Pediatric Endocrinology and Metabolism;2022-02-11

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