TGF-β/Smad3 Signaling Promotes Renal Fibrosis by Inhibiting miR-29-Reference-Cited by-同舟云学术

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TGF-β/Smad3 Signaling Promotes Renal Fibrosis by Inhibiting miR-29

Published:2011-07-22 Issue:8 Volume:22 Page:1462-1474
ISSN:1046-6673
Container-title:Journal of the American Society of Nephrology
language:en
Short-container-title:JASN

Author:

Qin Wei,Chung Arthur C.K.,Huang Xiao R.,Meng Xiao-Ming,Hui David S.C.,Yu Cheuk-Man,Sung Joseph J.Y.,Lan Hui Y.

Publisher

American Society of Nephrology (ASN)

Subject

Nephrology,General Medicine

Reference34 articles.

1. Smad7 as a therapeutic agent for chronic kidney diseases;Lan;Front Biosci,2008

2. Smad3 is key to TGF-β-mediated epithelial-to-mesenchymal transition, fibrosis, tumor suppression and metastasis

3. Essential Role of Smad3 in Angiotensin II–Induced Vascular Fibrosis

4. Essential role for Smad3 in angiotensin II-induced tubular epithelial-mesenchymal transition;Yang;J Pathol,2010

5. Advanced Glycation End-Products Induce Tubular CTGFviaTGF-β–Independent Smad3 Signaling

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