Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair In Vivo
Author:
Affiliation:
1. DNA Damage Tolerance CNRS, UMR7258, Marseille, France
2. Institut Paoli-Calmettes, Marseille, France
3. Aix-Marseille University, UM 105, Marseille, France
4. Inserm, U1068, CRCM, Marseille, France
Publisher
The Korean Society of Toxicology
Subject
Health, Toxicology and Mutagenesis,Toxicology
Link
http://pdf.medrang.co.kr/TR/2018/034/TR034-04-03.pdf
Reference46 articles.
1. Howard-Flanders, P, and Theriot, L (1966). Mutants of Escherichia coli K-12 defective in DNA repair and in genetic recombination. Genetics. 53, 1137-1150.
2. RecA-Dependent Recovery of Arrested DNA Replication Forks
3. Discontinuities in the DNA synthesized in an Excision-defective strain of Escherichia coli following ultraviolet irradiation
4. Replication fork stalling and cell cycle arrest in UV-irradiated Escherichia coli
5. Dynamics of Leading-Strand Lesion Skipping by the Replisome
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2. Rad51 filaments assembled in the absence of the complex formed by the Rad51 paralogs Rad55 and Rad57 are outcompeted by translesion DNA polymerases on UV-induced ssDNA gaps;PLOS Genetics;2023-02-07
3. REV1-Polζ maintains the viability of homologous recombination-deficient cancer cells through mutagenic repair of PRIMPOL-dependent ssDNA gaps;Molecular Cell;2021-10
4. The homologous recombination complex of the Rad51 paralogs Rad55-Rad57 avoids translesion DNA polymerase recruitment and counterbalances mutagenesis induced by UV radiation;2021-05-27
5. A Comprehensive View of Translesion Synthesis in Escherichia coli;Microbiology and Molecular Biology Reviews;2020-08-19
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